Can you explain:
1.How O2 and CO2 are carried by blood?
2.Why Emily can initially blow off excess CO2 (hyperventilating) but is still hypoxaemic?
3.Why she is still hypoxaemic despite inhaling supplemental oxygen?
4.Why her blood pH would be alkaline during one ABG analysis and acid during the other? Which is which?
5.How the oxyhaemoglobin curve would be shifted during different stages of the case?
6.How beta-agonists like salbutamol should relieve bronchoconstriction?
7.What would be different if Emily had blocked a main bronchus with an inhaled nut, rather than suffering from a severe asthma attack?
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